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According to our findings, NSS provided a novel and effective strategy for the prevention of MSG-induced renal damage especially on the levels of cytoarchitecture and programmed cell death before the renal pathogenic biomarkers can be altered

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None of them target the main (and unique) cause which is anoxia, or the main mechanism by which anoxia produces its effects, namely gluconeogenesis (Cori cycle)

The initial biochemical reaction involves the conversion of ethanol to acetaldehyde, which is primarily mediated by alcohol dehydrogenase and, to a lesser extent, by CYP2E1 and catalase [14]